Sendai virus - traducción al ruso
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Sendai virus - traducción al ruso

SPECIES OF VIRUS
Hemagglutinating virus of Japan; Hemagglutinating Virus of Japan; Sendai virus
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  • Infection spreads with variable efficiency. The virus was visualized with green fluorescent antibodies, and [[cell nuclei]] were stained with [[DAPI]] blue fluorescent dye. Photographs were taken immediately after the addition of virus to the cells and 26 hours later.
  • Sendai virus constructs that express luciferase. Upstream insertion of luciferase in rSeV-luc (P-M) resulted in greater luciferase activity than downstream insertion in rSeV-luc (F-HN).
  • [[Electron microscopy]] of virus
  • Schematic diagram of Sendai Virus genome with Green Fluorescent Protein (GFP).  (A) The recombinant virus genome expresses eGFP (239 residues) fused to the C-terminus of the L protein (2,228 residues). (B) Western blot analysis of GFP expressed in infected cells. Lysates of mock- (lane 1), recombinant virus genomes (lane 2 and lane 3) infected cells were reacted with anti-GFP antibody. (C) Analysis of construct localization in live cells. HeLa cells were infected with recombinant genomes and images of the cells were captured at the indicated hours post infection.
  • '''Genome structure'''<br/>
The positions of [[translation initiation]] sites for products of the alternative reading frame of the P-coding [[mRNA]]
  • GFP reporter gene is inserted between M and F genes
  • Possible]] model depicting formation of the viral assembly complex
  • Intrinsic anti-tumor and anti-[[angiogenic]] functions of type I [[interferon]]s
  • Sendai virus life cycle
  • '''Canine mast cell tumors treated with oncolytic Sendai virus.'''<br/>
Case 1. Male dog of 7 years old developed cutaneous, ulcerated, and poorly differentiated mastocytoma (35&nbsp;mm diameter) located close to his anus. (1) Primary tumor; (2) 2 weeks after the first virus treatment; (3) 4 weeks after the first virus treatment.<br/>
Case 2. Male German shorthaired pointer of 9 years old developed subcutaneous, regional (stage 2) intermediately differentiated mastocytoma. The primary tumor was removed without clean margins. (1) secondary growth 1 week after the surgical procedure; (2) 2 weeks after the first virus treatment; (3) 5 weeks after the first virus treatment.
  • Non-invasive [[bioluminescence imaging]] of infection in the [[respiratory tract]]s of living mice
  • Non-invasive [[bioluminescence imaging]] of Sendai virus infection in the [[respiratory tract]]s of living mice
  • Hypothetical]] fusion mechanism of viral and cell plasma membrane
  • SeV construct with a modified protease cleavage site in its fusion protein (F) gene was created. The image shows intratumoral and intra-organ spread of recombinant [[virion]]s ''in vivo'' in a murine model of [[hepatoma]] which has been [[xenograft]]ed.
  • [[Phylogenetic tree]]
  • '''SeV cell entry receptors.''' The names of receptors with known high binding affinity to the virus are marked with stars. The names of receptors that are overexpressed in some malignancies are in bold and underlined.
  • [[Schematic]] representation of [[virion]]
  • Virus partially or completely loses [[oncolytic]] activity after adapting to growth in [[cell culture]]s
  • Variable sensitivity of different cell lines to infection
  • Viral stimulation of RIG-1 and MDA-5 mediated IFN production
  • Trafficking of [[nucleocapsid]]s is mediated by [[intracellular vesicle]]s
  • Sendai virus green fluorescent protein infection of ovine cells. Fluorescence microscopy images of alveolar macrophages (A), blood-derived macrophages (B), and ovine skin fibroblasts (C) infected with Sendai virus vector expressing the GFP (right panel) at a multiplicity of infection (MOI) of 10. Bright-field images are shown in the left panel. The three cell types and all cells in the three cultures are GFP-positive. Ovine fibroblasts remained GFP-positive after 13 in vitro culture passages ((C), third image).
  • '''The process of fusion of the virus envelope with the cell membrane.'''

Sendai virus         

общая лексика

вирус Сендай

vesicular stomatitis         

медицина

везикулярный стоматит

respiratory syncytial virus         
  • RSV structure and genome organization. (a) ~150 nm RSV virion particle and (b) single-stranded negative-sense RNA genome consisting of 10 genes (NS1-NS2-N-P-M-SH-G-F-M2-L).
  • Schematic image of RSV life cycle
  • Filamentous RSV particles
  • False-color artistic rendering of RSV. G- glycoproteins are shown in light blue, with F-glycoproteins in orange
  • A phylogenetic tree of the pneumovirus & paramyxovirus families
  • X-ray of a child with RSV bronchiolitis showing the typical bilateral perihilar fullness
  • Video: Respiratory Syncytial Virus and Bronchiolitis
  • Lumen of an obstructed bronchiole containing cellular debris and aggregates of sloughed epithelial cells
  • Electron micrograph of RSV particle, which has variable shapes.
VIRAL PATHOGEN OF THE HUMAN RESPIRATORY TRACT
Respiratory Syncytial Virus; Respiratory syncytial virus infections; Respiratory syncytial virus, human; Rs virus; Respiratory syncytial; RSV infection; Respiratory syncytial viruses; RS virus; Human respiratory syncytial virus; Respiratory Syncytial Infection; F lipoprotein; Human orthopneumovirus; Respiratory synctitial virus; HRSV

медицина

респираторно-синцитиальный вирус

Definición

вирус кори
В. рода парамиксовирусов; возбудитель одноименной болезни человека.

Wikipedia

Murine respirovirus

Murine respirovirus, formerly Sendai virus (SeV) and previously also known as murine parainfluenza virus type 1 or hemagglutinating virus of Japan (HVJ), is an enveloped, 150-200 nm–diameter, negative sense, single-stranded RNA virus of the family Paramyxoviridae. It typically infects rodents and it is not pathogenic for humans or domestic animals

Sendai virus (SeV) is a member of the genus Respirovirus. The virus was isolated in the city of Sendai in Japan in the early 1950s. Since then, it has been actively used in research as a model pathogen. The virus is infectious for many cancer cell lines (see below), and has oncolytic properties demonstrated in animal models and in naturally-occurring cancers in animals. SeV's ability to fuse eukaryotic cells and to form syncytium was used to produce hybridoma cells capable of manufacturing monoclonal antibodies in large quantities.

Recent applications of SeV-based vectors include the reprogramming of somatic cells into induced pluripotent stem cells and vaccine creation. For vaccination purpose the Sendai virus-based constructs could be delivered in a form of nasal drops, which may be beneficial in inducing a mucosal immune response. SeV has several features that are important in a vector for a successful vaccine: the virus does not integrate into the host genome, it does not undergo genetic recombination, it replicates only in the cytoplasm without DNA intermediates or a nuclear phase and it is does not cause any disease in humans or domestic animals. Sendai virus is used as a backbone for vaccine development against Mycobacterium tuberculosis that causes tuberculosis, against HIV-1 that causes AIDS and against other viruses, including those that cause severe respiratory infections in children. The latter include Human Respiratory Syncytial Virus (HRSV), Human Metapneumovirus (HMPV) and Human Parainfluenza Viruses (HPIV).

The vaccine studies against M. tuberculosis, HMPV, HPIV1 and, HPIV2 are in the pre-clinical stage, against HRSV a phase I clinical trail has been completed. The phase I clinical studies of SeV-based vaccination were also completed for HPIV1. They were done in adults and in 3- to 6-year-old children. As a result of vaccination against HPIV1 a significant boost in virus-specific neutralizing antibodies was observed. A SeV-based vaccine development against HIV-1 has reached a phase II clinical trial. In Japan intranasal Sendai virus-based SARS-CoV-2 vaccine was created and tested in a mouse model.

¿Cómo se dice Sendai virus en Ruso? Traducción de &#39Sendai virus&#39 al Ruso